Author

M. Conenna

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

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Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Kumar· 1 shared D. Larrea· 1 shared E. Area-Gomez· 1 shared F. Bartolini· 1 shared G. J.-e. Shin· 1 shared L. Di Marcotullio· 1 shared M. E. Pero· 1 shared P. Infante· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperCell Biology

A. Kumar

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1 paper

Recent·Most cited

MFN2-dependent recruitment of ATAT1 coordinates mitochondria motility with alpha-tubulin acetylation and is disrupted in CMT2A

A. Kumar, D. Larrea, M. E. Pero, P. Infante, M. Conenna, G. J.-e. Shin, W. B. Grueber, L. Di Marcotullio, E. Area-Gomez, F. Bartolini

This paper shows that MFN2 recruits ATAT1 to mitochondria to regulate microtubule acetylation and mitochondria transport, and that CMT2A mutations disrupt this process leading to axonal degeneration.

bioRxiv · Mar 15, 2023 · PreprintCell Biology