Author

M. D. Schaid

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

D. C. Peter· 2 shared H. N. Wienkes· 2 shared M. E. Kimple· 2 shared R. J. Fenske· 2 shared A. Pennati· 1 shared A. Reuter· 1 shared J. Galipeau· 1 shared K. A. Carbajal· 1 shared

Activity by Year

2021-2021 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 24 mutual co-author links

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2 shared papersPhysiology

D. C. Peter

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2 papers

Recent·Most cited

Independent mechanisms underlie the protective effect of dietary polyunsaturated fatty acid supplementation and G&alphaz deficiency on the early type 1 diabetes phenotype of Non-obese diabetic (NOD) mice

R. J. Fenske, H. N. Wienkes, D. C. Peter, M. D. Schaid, A. Pennati, J. Galipeau, M. E. Kimple

This study shows that EPA-enriched diets and Gz deficiency both improve glucose tolerance in NOD mice but through distinct mechanisms.

bioRxiv · Mar 13, 2021 · PreprintPhysiology

The protection of Gαz-null NOD mice from hyperglycemia is sexually dimorphic and only partially β-cell autonomous

R. J. Fenske, D. C. Peter, H. N. Wienkes, M. D. Schaid, A. Reuter, K. A. Carbajal, M. E. Kimple

This study shows that Gαz loss in β-cells partially protects NOD mice from T1D-like hyperglycemia, with sex-specific differences in glucose tolerance and insulitis.

bioRxiv · Feb 8, 2021 · PreprintPhysiology