Author

D. C. Peter

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Physiology· 2 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

M. E. Kimple· 3 shared R. J. Fenske· 3 shared A. Reuter· 2 shared H. N. Wienkes· 2 shared M. D. Schaid· 2 shared A. L. Brill· 1 shared A. M. Weeks· 1 shared A. Pennati· 1 shared

Activity by Year

2021-2021 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersPhysiology

M. E. Kimple

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3 papers

Recent·Most cited

Independent mechanisms underlie the protective effect of dietary polyunsaturated fatty acid supplementation and G&alphaz deficiency on the early type 1 diabetes phenotype of Non-obese diabetic (NOD) mice

R. J. Fenske, H. N. Wienkes, D. C. Peter, M. D. Schaid, A. Pennati, J. Galipeau, M. E. Kimple

This study shows that EPA-enriched diets and Gz deficiency both improve glucose tolerance in NOD mice but through distinct mechanisms.

bioRxiv · Mar 13, 2021 · PreprintPhysiology

The protection of Gαz-null NOD mice from hyperglycemia is sexually dimorphic and only partially β-cell autonomous

R. J. Fenske, D. C. Peter, H. N. Wienkes, M. D. Schaid, A. Reuter, K. A. Carbajal, M. E. Kimple

This study shows that Gαz loss in β-cells partially protects NOD mice from T1D-like hyperglycemia, with sex-specific differences in glucose tolerance and insulitis.

bioRxiv · Feb 8, 2021 · PreprintPhysiology

Human islet expression levels of Prostaglandin E2 synthetic enzymes, but not prostaglandin EP3 receptor, are positively correlated with markers of β-cell function and mass in non-diabetic obesity

N. A. Truchan, R. J. Fenske, H. K. Sandhu, A. M. Weeks, C. Patibandla, B. Wancewicz, S. Pabich, A. Reuter, J. M. Harrington, A. L. Brill, D. C. Peter, R. Nall, M. Daniels, M. Punt, C. E. Kaiser, , M. E. Kimple

The paper examines the relationship between PGE2 production and β-cell function in non-diabetic obesity, finding that PGE2 synthetic enzymes correlate with β-cell mass and function, while EP3 receptor does not.

bioRxiv · Feb 3, 2021Pharmacology & Toxicology