Author

M. Fulde

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:bacteria (other)· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

K. Tedin· 2 shared B. Kuropka· 1 shared B. Loll· 1 shared C. Hamilton· 1 shared D. Ghilarov· 1 shared F. Ghaziasaeedi· 1 shared H. Antelmann· 1 shared J. G. Heddle· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersMicrobiology

K. Tedin

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2 papers

Recent·Most cited

Gene amplifications cause high-level resistance against albicidin in Gram-negative bacteria

M. Saathoff, S. Kosol, T. Semmler, S. A. Wolf, N. Dimos, J. Kupke, M. Seidel, F. Ghaziasaeedi, K. Tedin, B. Kuropka, W. Czyszczon, D. Ghilarov, S. Graetz, J. G. Heddle, B. Loll, , M. Fulde

This study identifies gene amplification as a mechanism conferring high-level resistance to albicidin in Gram-negative bacteria, validated across multiple species.

bioRxiv · Sep 15, 2022Microbiology

Staphylococcus aureus uses the bacilliredoxin (BrxAB)/bacillithiol disulfide reductase (YpdA) redox pathway to defend against oxidative stress under infections

N. Linzner, V. V. Loi, V. N. Fritsch, Q. N. Tung, S. Stenzel, M. Wirtz, R. Hell, C. Hamilton, K. Tedin, M. Fulde, H. Antelmann

This study identifies the BrxAB/BSH/YpdA pathway as critical for S. aureus to manage oxidative stress and survive infections by maintaining BSH redox homeostasis.

bioRxiv · May 1, 2019Microbiology