Author

M. Pesic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Organism:Drosophila· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

V. M. Perez-Garcia· 2 shared A. C. Jacomin· 1 shared A. Orian· 1 shared A. Podolski-Renic· 1 shared A. Wiegering· 1 shared B. Segura-Collar· 1 shared C. Ade· 1 shared C. Fuss· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCancer Biology

V. M. Perez-Garcia

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Stabilisation of β-Catenin-WNT signalling by USP10 in APC-truncated colorectal cancer drives cancer stemness and enables super-competitor signalling

M. Reissland, O. Hartmann, S. Tauch, C. Prieto-Garcia, C. Schulte, D. Solvie, S. Loebbert, A. C. Jacomin, M. Pesic, J. M. Bugter, C. Schuelein-Voelk, C. Fuss, N. Pahor, C. Ade, V. Buck, , M. E. Diefenbacher

This study identifies USP10 as a deubiquitylase that stabilizes β-Catenin in APC-truncated colorectal cancer, enhancing WNT signaling and cancer stemness.

bioRxiv · Feb 10, 2023 · PreprintCancer Biology

On optimal temozolomide scheduling for slowly growing gliomas

B. Segura-Collar, J. Jimenez-Sanchez, R. Gargini, M. Dragoj, J. M. Sepulveda, M. Pesic, P. Sanchez-Gomez, V. M. Perez-Garcia

This study uses in-silico models and experimental data to show that longer TMZ dose intervals improve survival and reduce toxicity in slowly growing gliomas by altering tumor phenotype.

bioRxiv · Mar 10, 2022 · PreprintCancer Biology

Stochastic fluctuations drive non-genetic evolution of proliferation in clonal cancer cell populations

C. Ortega-Sabater, G. Fernandez-Calvo, J. Dinic, A. Podolski-Renic, M. Pesic, V. M. Perez-Garcia

The paper investigates how stochastic fluctuations in phenotypic traits drive non-genetic proliferation in clonal cancer cell populations, using mathematical models and experimental data.

bioRxiv · Jun 8, 2021Cancer Biology