Author

M. Uhrig

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:statistical model· 1

Frequent Co-Authors

B. S. Casas· 1 shared C. Chacon· 1 shared C. P. Prieto· 1 shared C. Wiese· 1 shared F. Ezquer· 1 shared G. Vitoria· 1 shared M. Casas· 1 shared M. Ezquer· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

B. S. Casas

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2 papers

Recent·Most cited

Schizophrenia-derived hiPSC brain microvascular endothelial cells show impairments in angiogenesis and blood-brain barrier function

B. S. Casas, G. Vitoria, C. P. Prieto, M. Casas, C. Chacon, M. Uhrig, F. Ezquer, M. Ezquer, S. K. Rehen, V. Palma

This study shows that schizophrenia-derived brain microvascular endothelial cells have impaired angiogenesis and blood-brain barrier function, suggesting a developmental origin of the disease.

bioRxiv · Apr 14, 2022Cell Biology

RAD51AP1 and RAD54 underpin two distinct RAD51-dependent routes of DNA damage repair via homologous recombination

P. Selemenakis, N. Sharma, Y. Kwon, M. Uhrig, P. Sung, C. Wiese

This study identifies RAD51AP1 and RAD54 as two distinct RAD51-dependent HR sub-pathways, showing their combined inactivation enhances cancer cell sensitivity to DNA damage agents.

bioRxiv · Jul 15, 2021 · PreprintCell Biology