Author

Michael Schneider

University Hospital Schleswig-HolsteinPrimary ORCID profile (0000-0002-4969-2218 · 3 papers)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Michael SchneideriD3 papers · Helmholtz Munich

Biology Disciplines

Click to filter author papers

Immunology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Assay:metabolomics· 1

Frequent Co-Authors

Carolin GragolliD· 2 shared Elias RawishiD· 2 shared Ingo Eitel· 2 shared Jorge Duque Escobar· 2 shared Katharina KurziD· 2 shared Tanja ZelleriD· 2 shared Thomas StiermaieriD· 2 shared Timo RusackiD· 2 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

Carolin GragollORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

C3a receptor signalling in ObRb-expressing neurons promotes leptin resistance and obesity-associated cardiac remodelling

Elias Rawish, Carolin Gragoll, Michael Schneider, Timo Rusack, Katharina Kurz, Violetta Pilorz, Sophia Marie Reiners, Sven Geisler, Janti HajAhmad, Nuria López‐Alcántara, Nada Alnaggar, Andreas Teufel, Mirko Otto, Jorge Duque Escobar, Thomas Stiermaier, , Ingo Eitel

This study identifies C3aR signaling in ObRb-expressing neurons as a pathway contributing to leptin resistance and obesity-related cardiac remodeling, with potential therapeutic implications.

bioRxiv · Oct 7, 2026 · PreprintImmunology

Sphingolipid-Driven Lipotoxicity Promotes Calcific Aortic Valve Disease Through ER-Stress-NF-κB-EndMT Signaling and Is Therapeutically Targetable Through Serine Palmitoyltransferase Inhibition

Elias Rawish, Timo Rusack, Peter Ruben Letsch, Julica Inderhees, Kristian Haendler, Carolin Gragoll, Jakob Christoph Voran, Michael Schneider, Katharina Kurz, Mohammed Rabiul Hosen, Maren Behrensen, Svenja Denker, Axel Kuenstner, Thorben Sauer, Jorge Duque Escobar, , Christian Frerker

This study identifies ceramide-driven lipotoxicity as a driver of calcific aortic valve disease and shows that SPT inhibition can reverse disease progression in vivo.

bioRxiv · Oct 7, 2026 · PreprintMolecular Biology