Author

N. Araki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Nishigaki· 1 shared B. Baron· 1 shared D. Kobayashi· 1 shared D. Paudel· 1 shared H. Okita· 1 shared K. Kawai· 1 shared M. Furutani-Seiki· 1 shared M. Kobayashi· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCell Biology

A. Nishigaki

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

AT-hook DNA-binding motif-containing protein one knockdown downregulates EWS-FLI1 transcriptional activity in Ewings sarcoma cells

T. Kitagawa, D. Kobayashi, B. Baron, H. Okita, T. Miyamoto, R. Takai, D. Paudel, T. Ohta, Y. Asaoka, M. Tokunaga, M. Furutani-Seiki, N. Araki, Y. Kuramitsu, M. Kobayashi

The paper identifies AHDC1 as a protein interacting with EWS-FLI1 in Ewing's sarcoma and shows its knockdown reduces cell growth and transcriptional activity.

bioRxiv · May 16, 2022 · PreprintCancer Biology

Rab10-positive tubular structures represent a novel endocytic pathway that diverges from canonical macropinocytosis in RAW264 macrophages

K. Kawai, A. Nishigaki, S. Moriya, Y. Egami, N. Araki

This study identifies Rab10-positive tubules as a novel endocytic pathway in macrophages that diverges from canonical macropinocytosis and does not involve lysosomal degradation.

bioRxiv · Jan 3, 2021Cell Biology