Author

N. Guiraud

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:proteomics· 1

Frequent Co-Authors

c. recher· 2 shared E. Saland· 2 shared J.-E. Sarry· 2 shared A. Sahal· 1 shared C. Bosc· 1 shared C. Larrue· 1 shared C. Schliemann· 1 shared d. fesquet· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

c. recher

Mutual co-authors in cluster

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2 papers

Recent·Most cited

SHED-dependent oncogenic signalling of the PEAK3 pseudo-kinase

y. OUNOUGHENE, E. fourgous, Y. Boublik, E. Saland, N. Guiraud, c. recher, s. urbach, P. P. Fort, J.-E. Sarry, d. fesquet, s. roche

This study identifies PEAK3 as an oncogenic pseudo-kinase that uses the SHED module to activate AKT signaling and promote cancer growth.

bioRxiv · Aug 30, 2021 · PreprintCancer Biology

Adrenomedullin-CALCRL Axis Controls Relapse-Initiating Drug Tolerant Acute Myeloid Leukemia Cells

C. Larrue, N. Guiraud, P.-L. Mouchel, M. Dubois, T. Farge, M. Gotanegre, C. Bosc, E. Saland, M.-L. Travers-Nicolau, M. Sabatier, N. Serhan, A. Sahal, E. Boet, S. Mouche, Q. Heydt, , J.-E. Sarry

The paper identifies the ADM-CALCRL axis as a driver of drug-tolerant AML cells that persist after chemotherapy and proposes it as a therapeutic target to prevent relapse.

bioRxiv · Aug 17, 2020Cancer Biology