Author

Y. Boublik

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Approach:biophysical modeling· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

E. Fourgous· 2 shared S. Roche· 2 shared A. Fernandez· 1 shared A.-L. Le Roux· 1 shared c. recher· 1 shared d. fesquet· 1 shared E. Saland· 1 shared I.-L. Mohammad· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersBiophysics

E. Fourgous

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Author profile →

2 papers

Recent·Most cited

Lipid-mediated dimerization of membrane-anchored c-Src is driven by a cluster of lysine residues in the N-terminal SH4 domain.

I.-L. Mohammad, J. Carvajal, A. Fernandez, M. Taules, E. Fourgous, Y. Boublik, A.-L. Le Roux, S. Roche, M. Pons

This study identifies a cluster of lysine residues in the SH4 domain of c-Src that enhances lipid-mediated dimerization, increasing tyrosine phosphorylation in HEK293T cells.

bioRxiv · May 31, 2022 · PreprintBiophysics

SHED-dependent oncogenic signalling of the PEAK3 pseudo-kinase

y. OUNOUGHENE, E. fourgous, Y. Boublik, E. Saland, N. Guiraud, c. recher, s. urbach, P. P. Fort, J.-E. Sarry, d. fesquet, s. roche

This study identifies PEAK3 as an oncogenic pseudo-kinase that uses the SHED module to activate AKT signaling and promote cancer growth.

bioRxiv · Aug 30, 2021 · PreprintCancer Biology