Author

Olivia Baumberger

ETH Zurich

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Amro A. HussieniD· 1 shared Archana G. MohananiD· 1 shared Barbara P. Niederöst· 1 shared Bart GhesquièreiD· 1 shared Diane E. TayloriD· 1 shared Didier SurdeziD· 1 shared Dirk Sw ElewautiD· 1 shared Evi MasscheleiniD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

Amro A. HussienORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

HIF1α gates tendon response to overload and drives tendinopathy independently of vascular recruitment

Greta Moschini, Archana G. Mohanan, Izabella S. Niewczas, Diane E. Taylor, Patrick Klaus Jaeger, Guillermo Turiel, Amro A. Hussien, Stefania L. Wunderli, Olivia Baumberger, Maja Wolleb, Florence Marti, Barbara P. Niederöst, Maja Bollhalder, Raphaela Ardicoglu, Nathalie Tisch, , Jess Gerrit Snedeker

Mechanical overload activates hypoxia-inducible factor 1-alpha (HIF1a) in tenocytes to drive tendinopathy independently of VEGFA-mediated vascular ingrowth, and tenocyte Hif1a deletion prevents overload-induced tendon degeneration in mice.

Science Translational Medicine · Jan 7, 2026Peer-reviewedCell Biology✦ In-depth digest