Author

P. Pecina

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:metabolism & bioenergetics· 1 Organism:rat· 1

Frequent Co-Authors

J. Houstek· 3 shared M. Vrbacky· 3 shared T. Mracek· 3 shared A. Pecinova· 2 shared K. Cunatova· 2 shared K. Tauchmannova· 2 shared V. Kaplanova· 2 shared D. Pajuelo Reguera· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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3 shared papersBiochemistry

J. Houstek

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Physiological variability in mitochondrial rRNA predisposes to metabolic syndrome

P. Pecina, K. Cunatova, V. Kaplanova, G. Puertas-Frias, J. Silhavy, K. Tauchmannova, M. Vrbacky, T. Cajka, O. Gahura, M. Pravenec, J. Houstek, T. Mracek, A. Pecinova

The paper investigates how mitochondrial rRNA sequence variation contributes to insulin resistance and metabolic syndrome in rats fed a high-fat diet.

bioRxiv · Nov 8, 2023Physiology

MLQ is responsible for stabilisation of subunit a in the holoenzyme of mammalian ATP synthase

K. Tauchmannova, H. D. Ho, H. Nuskova, A. Pecinova, L. Alan, M. Rodinova, E. Konarikova, M. Vrbacky, G. Puertas, V. Kaplanova, J. Houstek, P. Pecina, T. Mracek

This paper investigates the role of MLQ in ATP synthase biogenesis, finding that MLQ stabilizes the holoenzyme and its absence leads to labile, functional but ATP-synthesizing-defective complexes.

bioRxiv · Feb 3, 2020 · PreprintBiochemistry

Loss of COX4i1 leads to combined respiratory chain deficiency and impaired mitochondrial proteosynthesis

K. Cunatova, D. Pajuelo Reguera, M. Vrbacky, E. Fernandez-Vizarra, S. Ding, I. Fearnley, M. Zeviani, J. Houstek, T. Mracek, P. Pecina

This study shows that loss of COX4i1 causes combined respiratory chain deficiency and impaired mitochondrial proteosynthesis by disrupting cI and cIV biogenesis.

bioRxiv · Jan 31, 2020 · PreprintBiochemistry