Author

S. E. Scheuermann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:mouse· 1 Organism:non-human primate· 1

Frequent Co-Authors

B. J. Beddingfield· 2 shared L. A. Rowe· 2 shared N. J. Maness· 2 shared A. Melton· 1 shared B. Smith· 1 shared C. Krzykwa· 1 shared F. Schiro· 1 shared G. M. Reyes· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersImmunology

B. J. Beddingfield

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The Impact of SIV-Induced Immunodeficiency on Clinical Manifestation, Immune Response, and Viral Dynamics in SARS-CoV-2 Coinfection

A. Melton, L. A. Rowe, T. Penney, C. Krzykwa, K. Goff, S. E. Scheuermann, H. J. Melton, K. Williams, N. Golden, K. Moore Green, B. Smith, K. Russell-Lodrigue, J. P. Dufour, L. A. Doyle-Meyers, F. Schiro, , N. J. Maness

This study examines how SIV-induced immunodeficiency affects SARS-CoV-2 infection in macaques, finding that while immune responses are impaired, viral kinetics and evolution remain unchanged.

bioRxiv · Nov 15, 2023 · PreprintImmunology

Real time analysis of SARS-CoV-2 induced cytolysis reveals distinct variant-specific replication profiles

S. E. Scheuermann, K. A. Goff, L. A. Rowe, B. J. Beddingfield, N. J. Maness

This study uses real-time cell analysis to compare replication kinetics of SARS-CoV-2 variants, finding distinct replication profiles among WA1/2020, Delta, and Omicron subvariants.

bioRxiv · Mar 28, 2023 · PreprintMicrobiology

The Histoplasma capsulatum DDR48 Gene Is Required For Survival Within Macrophages, Response To Oxidative Stress, And Resistance to Antifungal Drugs

L. T. Blancett, K. A. Runge, G. M. Reyes, L. A. Kennedy, S. C. Jackson, S. E. Scheuermann, M. B. Harmon, J. C. Williams, G. Shearer

This paper identifies HcDDR48 as essential for Histoplasma capsulatum survival in macrophages, oxidative stress response, and antifungal drug resistance.

bioRxiv · Oct 25, 2020 · PreprintMicrobiology