Author

S. Pankow

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:experimental assay· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

J. K. Diedrich· 2 shared J. R. Yates· 2 shared A. A. Wilson· 1 shared C. Torres-Arancivia· 1 shared D. C. Liberti· 1 shared D. N. Kotton· 1 shared G. J. Murphy· 1 shared J. D. Rosarda· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersBiochemistry

J. K. Diedrich

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Author profile →

2 papers

Recent·Most cited

Covalent Protein Painting Reveals Structural Changes in the Proteome in Alzheimer Disease

T. C. Bamberger, S. Pankow, S. Martinez-Bartolome, M. Ma, J. K. Diedrich, R. A. Rissman, J. R. Yates

The paper introduces CPP, a method to quantify lysine accessibility in proteins, and finds altered lysine accessibility in Alzheimer's disease patients.

bioRxiv · Jan 31, 2020Biochemistry

Amyloidogenic Proteins Drive Hepatic Proteostasis Remodeling in an Induced Pluripotent Stem Cell Model of Systemic Amyloid Disease

R. M. Giadone, D. C. Liberti, T. M. Matte, J. D. Rosarda, C. Torres-Arancivia, S. Ghosh, J. K. Diedrich, S. Pankow, N. Skvir, J.-C. Jean, J. R. Yates, A. A. Wilson, L. H. Connors, D. N. Kotton, R. L. Wiseman, G. J. Murphy

This study uses iPSC-derived hepatic cells to show that liver proteostasis factors and UPR signaling correlate with TTR amyloid production and can reduce amyloid secretion through ER stress-independent activation.

bioRxiv · Nov 10, 2019 · PreprintCell Biology