Author

S. Timonen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:drug response / drug discovery· 2 Organism:human· 2 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

K. Wennerberg· 2 shared T. Aittokallio· 2 shared A. Jaiswal· 1 shared D. Bulanova· 1 shared E. A. Pietila· 1 shared J. Hynninen· 1 shared J. Oikkonen· 1 shared K. Lehti· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCancer Biology

K. Wennerberg

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2 papers

Recent·Most cited

A synthetic lethal dependency on casein kinase 2 in response to replication-perturbing drugs in RB1-deficient ovarian and breast cancer cells

D. Bulanova, Y. Akimov, W. Senkowski, J. Oikkonen, L. Gall-Mas, S. Timonen, M. Elmadani, J. Hynninen, S. Hautaniemi, T. Aittokallio, K. Wennerberg

This study identifies CK2 inhibition as a sensitizing strategy for RB1-deficient ovarian and breast cancer cells to replication-perturbing drugs, showing enhanced efficacy when combined with existing therapies.

bioRxiv · Nov 14, 2022 · PreprintCancer Biology

Multi-modal meta-analysis of cancer cell line omics profiles identifies ECHDC1 as a novel breast tumor suppressor

A. Jaiswal, P. Gautam, E. A. Pietila, S. Timonen, N. Nordstrom, Z. Tanoli, N. Sipari, K. Lehti, K. Wennerberg, T. Aittokallio

The paper integrates multi-modal omics data from cancer cell lines to identify ECHDC1 as a novel breast tumor suppressor and discover synthetic lethal interactions.

bioRxiv · Jan 31, 2020Cancer Biology