Author

S. Treves

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:drug response / drug discovery· 1 Assay:imaging· 1

Frequent Co-Authors

A. Ruiz· 2 shared F. Zorzato· 2 shared A. Schmidt· 1 shared C. P. Bachmann· 1 shared F. Noreen· 1 shared F. Protasi· 1 shared J. Eckhardt· 1 shared L. Pietrangelo· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersPhysiology

A. Ruiz

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2 papers

Recent·Most cited

Quantitative proteomic analysis of skeletal muscles from wild type and transgenic mice carrying recessive Ryr1 mutations linked to congenital myopathies

J. Eckhardt, A. Ruiz, S. Koenig, M. Frieden, A. Schmidt, S. Treves, F. Zorzato

The paper quantitatively analyzes proteomic differences in skeletal muscles of wild type and Ryr1 mutant mice, revealing reduced RyR1 and altered protein content linked to congenital myopathies.

bioRxiv · Sep 26, 2022Physiology

Improvement of muscle strength in a mouse model for congenital myopathy treated with HDAC and DNA methyltransferase inhibitors

A. Ruiz, S. Benucci, U. Duthaler, C. P. Bachmann, M. Franchini, F. Noreen, L. Pietrangelo, F. Protasi, S. Treves, F. Zorzato

This study treats a mouse model of congenital myopathy with HDAC and DNA methyltransferase inhibitors, improving muscle strength and RyR1 protein content.

bioRxiv · Nov 9, 2021Physiology