Author

Siri L. Peter

University of Zurich

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:deep learning· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Chantal Bodenmann· 3 shared Christian TackenbergiD· 3 shared Beatriz Achón BuiliD· 2 shared Kathrin J. Zürcher· 2 shared Melanie GeneraliiD· 2 shared Nora H. RentschiD· 2 shared R. WeberiD· 2 shared Ruslan RustiD· 2 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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3 shared papersNeuroscience

Chantal Bodenmann

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Neural graft elimination via dual safety switch compromises therapeutic recovery after stroke

Beatriz Achón Buil, Nora H. Rentsch, R. Weber, Carmen Helfenstein, Chantal Bodenmann, Kathrin J. Zürcher, Siri L. Peter, Melanie Generali, Christian Tackenberg, Ruslan Rust

This study shows that using a dual safety switch to eliminate neural grafts after stroke impairs functional recovery despite effective cell ablation.

bioRxiv · Jun 2, 2026 · PreprintNeuroscience

Neural xenografts contribute to long-term recovery in stroke via molecular graft-host crosstalk

R. Weber, Beatriz Achón Buil, Nora H. Rentsch, Patrick Perron, Stefanie Halliday, Allison Bosworth, Mingzi Zhang, Kassandra Kisler, Chantal Bodenmann, Kathrin J. Zürcher, Daniela Uhr, Debora Meier, Siri L. Peter, Melanie Generali, Shuo Lin, , Ruslan Rust

This study shows that transplanting iPSC-derived neural progenitor cells improves long-term recovery after stroke in mice by promoting tissue repair and reducing neurological deficits.

Nature Communications · Sep 16, 2025Peer-reviewedNeuroscience

APOE4 increases energy metabolism in APOE-isogenic iPSC-derived neurons

Vanessa Budny, Yannic Knöpfli, Deborah Meier, Kathrin Zürcher, Chantal Bodenmann, Siri L. Peter, Terry Müller, Marie Tardy, Cédric Cortijo, Christian Tackenberg

This study shows APOE4 increases mitochondrial ATP production in neurons compared to other APOE isoforms, suggesting a gain-of-function effect on neuronal energy metabolism.

bioRxiv · Jun 3, 2024Neuroscience