Author

Stefano Rossi

University of ManchesterPrimary ORCID profile (0000-0003-0006-7013 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Stefano RossiiD1 paper · Università degli Studi della Tuscia

Biology Disciplines

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Genetic & Genomic Medicine· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:ATAC-seq· 1 Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1 Organism:mouse· 1

Frequent Co-Authors

Alexander Froese· 1 shared Antonios FrantzeskosiD· 1 shared Bing Qi· 1 shared Chenfu ShiiD· 1 shared Cristina E. Molina· 1 shared Danyun ZhaoiD· 1 shared David Revuelta· 1 shared Gisela OrozcoiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPharmacology & Toxicology

Alexander Froese

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Author profile →

2 papers

Recent·Most cited

Submembrane geography dictates GPCR signalling compartmentation and physiological function

Alexander Froese, Bing Qi, Miao Feng, Greta Boeer, Karoline Morhenn, Svenja Kupsch, Xing Sun, Hariharan Subramanian, Jose L. Sanchez-Alonso, Roman Medvedev, Peter T. Wright, Mart Gallego, David Revuelta, Nadja I. Beck, Cristina E. Molina, , Viacheslav Nikolaev

This study shows that GPCR signaling varies based on submembrane location, with different functional outcomes in cardiomyocytes, and introduces submembrane-specific beta-blockers that target specific nanodomains.

bioRxiv · Nov 3, 2026 · PreprintPharmacology & Toxicology

Identification of causal genes and mechanisms by which genetic variation mediates juvenile idiopathic arthritis susceptibility using functional genomics and CRISPR-Cas9

Antonios Frantzeskos, Valeriya Malysheva, Chenfu Shi, Danyun Zhao, Muskan Gupta, Stefano Rossi, James Ding, Wendy Thomson, Stephen Eyre, John Bowes, Mikhail Spivakov, Gisela Orozco

This study identifies causal genes and mechanisms for juvenile idiopathic arthritis by integrating GWAS, PCHi-C, ATAC-seq, and CRISPRa/i in CD4+ T cells from JIA patients.

medRxiv · May 22, 2025 · PreprintGenetic & Genomic Medicine