Author

T. Jimenez

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Molero· 2 shared G. Serrano· 2 shared J. Montoro· 2 shared M. Ainciburu· 2 shared A. Alfonso Pierola· 1 shared A. Alfonso-Pierola· 1 shared A. Diaz Mazkiaran· 1 shared A. Diaz-Mazkiaran· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCancer Biology

A. Molero

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Single cell profiling of del(5q) MDS unveils its transcriptional landscape and the impact of lenalidomide

G. Serrano, N. Berastegui Zufiaurre, A. Diaz Mazkiaran, P. Garcia Olloqui, S. Huerga Dominguez, A. Alfonso Pierola, A. Ullate Agote, B. Ariceta Ganuza, M. Ainciburu, A. Vilas Zornoza, P. San Martin Uriz, P. Aguirre Ruiz, J. M. Lamo de Espinosa, P. Acha, O. Calvete, , M. Hernaez Arrazola

This study uses scRNA-seq to analyze del(5q) MDS patients, revealing shared and unique transcriptional alterations and lenalidomide's impact on disease and treatment response.

bioRxiv · Oct 19, 2023 · PreprintCancer Biology

Transcriptional regulation of HSCs in Aging and MDS reveals DDIT3 as a Potential Driver of Dyserythropoiesis

N. Berastegui, M. Ainciburu, J. P. Romero, A. Alfonso-Pierola, C. Philippe, A. Vilas-Zornoza, P. San Martin, R. Ordonez, D. O. Alignani, S. Sarvide, L. Castro, J. M. Lamo-Espinosa, M. San-Julian, T. Jimenez, F. Lopez Cardenas, , F. Prosper

This study identifies DDIT3 as a driver of dyserythropoiesis in MDS by showing its upregulation in HSCs and its ability to disrupt erythropoiesis when overexpressed.

bioRxiv · Sep 8, 2021 · PreprintCancer Biology