Author

T. Kiyoi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

H. Mori· 1 shared H. Nakazawa· 1 shared I. Muramatsu· 1 shared J. Muto· 1 shared J. Uwada· 1 shared K. Sayama· 1 shared K. Shiraishi· 1 shared K. Watanabe· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

H. Mori

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2 papers

Recent·Most cited

PIKFYVE inhibition induces endosome- and lysosome-derived vacuole enlargement via ammonium accumulation

J. Uwada, H. Nakazawa, T. Kiyoi, T. Yazawa, I. Muramatsu, T. Masuoka

This study shows that PIKFYVE inhibition causes endosome and lysosome enlargement through ammonium accumulation from glutamine metabolism, impairing lysosomal function and autophagic flux.

bioRxiv · Apr 19, 2024Cell Biology

Highly concentrated trehalose induces transient senescence-associated secretory phenotype in fibroblasts via CDKN1A/p21

J. Muto, S. Fukuda, K. Watanabe, X. Dai, T. Tsuda, T. Kiyoi, H. Mori, K. Shiraishi, M. Murakami, S. Higashiyama, Y. Mizukami, K. Sayama

High-concentration trehalose induces a beneficial senescence-associated secretory phenotype in fibroblasts via CDKN1A/p21, enhancing wound repair in mice.

bioRxiv · Oct 6, 2021 · PreprintCell Biology