Author

T. Repasy

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

J. R. Johnson· 3 shared J. S. Cox· 3 shared N. J. Krogan· 3 shared D. A. Portnoy· 2 shared J. M. Budzik· 2 shared A. W. Roberts· 1 shared A. Y. Lee· 1 shared B. H. Penn· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersMicrobiology

J. R. Johnson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Deficiency in Galectin-3, -8, and -9 impairs immunity to chronic Mycobacterium tuberculosis infection but not acute infection with multiple intracellular pathogens

H. M. Morrison, J. Craft, R. Rivera-Lugo, J. R. Johnson, G. R. Golovkine, C. E. Dodd, E. Van Dis, W. L. Beatty, S. R. Margolis, T. Repasy, I. Shaker, A. Y. Lee, R. E. Vance, S. A. Stanley, N. J. Krogan, , J. S. Cox

bioRxiv · Dec 29, 2022 · PreprintImmunology

Comparative analysis of macrophage post-translational modifications during intracellular bacterial pathogen infection

J. R. Johnson, T. Parry, T. Repasy, K. M. Geiger, E. Verschueren, J. M. Budzik, D. Jimenez-Morales, B. W. Newton, E. Powell, L. Coscoy, D. A. Portnoy, N. J. Krogan, J. S. Cox

This study uses proteomics to analyze post-translational modifications in macrophages infected with three intracellular pathogens, revealing pathogen-specific molecular responses and a novel mechanism of virus-bacterial synergy.

bioRxiv · May 27, 2020 · PreprintMicrobiology

Global proteomic profiling of primary macrophages during M. tuberculosis infection identifies TAX1BP1 as a mediator of autophagy targeting

J. M. Budzik, N. E. Garelis, T. Repasy, A. W. Roberts, L. Popov, T. J. Parry, D. Jiminez-Morales, D. Swaney, J. R. Johnson, N. J. Krogan, J. S. Cox

The paper uses proteomic analysis to identify TAX1BP1 as a mediator of autophagy targeting M. tuberculosis during macrophage infection.

bioRxiv · Jan 31, 2019 · PreprintMicrobiology