Author

V. B. Sreenu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Microbiology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Approach:statistical model· 2 Research Focus:differential expression· 1 Research Focus:phylogenetics / evolution· 1 Assay:proteomics· 1

Frequent Co-Authors

A. da Silva Filipe· 2 shared A. Wickenhagen· 2 shared J. Hughes· 2 shared L. Tong· 2 shared A. Arossa· 1 shared A. Ceschi· 1 shared A. De Marco· 1 shared A. Dhingra· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersMicrobiology

A. da Silva Filipe

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The Interferon Resistance of Transmitted HIV-1 is a Consequence of Enhanced Replicative Fitness

E. Sugrue, A. Wickenhagen, N. Mollentze, M. A. Aziz, V. B. Sreenu, S. Truxa, L. Tong, A. da Silva Filipe, D. L. Robertson, J. Hughes, S. J. Rihn, S. J. Wilson

The paper identifies that transmitted HIV-1's interferon resistance stems from enhanced replicative fitness rather than specific IFN resistance.

bioRxiv · Dec 18, 2021Microbiology

The circulating SARS-CoV-2 spike variant N439K maintains fitness while evading antibody-mediated immunity

E. C. Thomson, L. E. Rosen, J. G. Shepherd, R. Spreafico, A. da Silva Filipe, J. A. Wojcechowskyj, C. Davis, L. Piccoli, D. J. Pascall, J. Dillen, S. Lytras, N. Czudnochowski, R. Shah, M. Meury, N. Jesudason, , J. Nichols

The paper shows the N439K SARS-CoV-2 spike variant maintains replication fitness while evading antibody neutralization.

bioRxiv · Nov 4, 2020Microbiology

Human Cytomegalovirus Genomes Sequenced Directly from Clinical Material: Variation, Multiple-Strain Infection, Recombination and Mutation

N. M. Suarez, G. S. Wilkie, E. Hage, S. Camiolo, M. Holton, J. Hughes, M. Maabar, V. B. Sreenu, A. Dhingra, U. A. Gompels, G. W. Wilkinson, F. Baldanti, F. Milena, D. Lilleri, A. Arossa, , A. J. Davison

The paper sequences HCMV genomes from clinical samples to study variation, multiple-strain infection, recombination, and mutation, finding that recombinant strains and pseudogenes can persist and cause congenital infections.

bioRxiv · Feb 18, 2019Microbiology