Author

Vindhya Chaganty

Agency for Science, Technology and Research

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetic & Genomic Medicine· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:statistical model· 2 Organism:zebrafish· 2 Assay:bulk RNA-seq· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Ain Nur Ali· 1 shared Ajay Sriram MathuruiD· 1 shared Alex N. BullockiD· 1 shared Alvin Yu Jin NgiD· 1 shared Anand Kumar AndiappaniD· 1 shared Birsen KaramaniD· 1 shared Bruno ReversadeiD· 1 shared Byrappa VenkateshiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperGenetic & Genomic Medicine

Ain Nur Ali

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2 papers

Recent·Most cited

Comparative transcriptomics of lateral hypothalamic cell types reveals conserved growth hormone-tachykinin dynamics in feeding

Vindhya Chaganty, Ruey‐Kuang Cheng, Kimberle Shen, Na Zhang, Gaynah Javier Doblado, Sarah Ong, Sandra Tan, Valarie Yu Yan Tham, Jung-Hwa Choi, Marnie E. Halpern, Wei Leong Chew, Anand Kumar Andiappan, Sarah Xinwei Luo, Caroline Lei Wee

The paper identifies conserved GABAergic neurons in zebrafish and mouse lateral hypothalamus that respond to growth hormone and food cues, revealing a shared mechanism for appetite regulation.

Molecular Metabolism · Sep 13, 2025Peer-reviewedNeuroscience

Loss of IVNS1ABP, a gigaxonin paralogue, leads to a progeroid neuropathy due to impaired proteostasis

Carine Bonnard, Ain Nur Ali, Yuan Fang, Kiat Y. Tan, Yan T. Lim, Puck Wee Chan, Nardev Ramanathan, Nur A.B.M. Kamsani, Vindhya Chaganty, Ding Xiong, Ye Sing Tan, Shu-Min Chou, Marwa Chourabi, Gunaseelan Narayanan, Alvin Yu Jin Ng, , Bruno Reversade

This study shows that loss of IVNS1ABP causes a progeroid neuropathy by impairing proteostasis, leading to protein aggregation and lysosomal dysfunction in neuronal cells.

medRxiv · Oct 23, 2024 · PreprintGenetic & Genomic Medicine