Dillon C. Gavlock

This paper develops a coupled microphysiological system to study how liver dysfunction affects pancreatic islet function, finding altered secretomes and glucose responses under metabolic syndrome conditions.

This study uses a human liver microphysiology system to compare the effects of PNPLA3 variants on MASLD progression and drug response.

This study creates a human iPSC-derived hepatocyte model with the TM6SF2 E167K mutation to study its effects on lipid metabolism and MASLD.