Amílcar Tanuri

This study shows that Zika virus reprograms the glioblastoma tumor microenvironment to be anti-tumoral by inducing type III interferon signaling, which can be mimicked by exogenous treatment.

This study shows that Oropouche virus strains cause microcephaly-like effects in brain organoids by disrupting neural development and inducing cell death and apoptosis.

This study compares replication capacity and nucleocapsid proteoforms across SARS-CoV-2 variants in human pulmonary cells, finding Omicron BA.1 has reduced replication and variant-specific proteoforms.

The paper describes an outbreak of toxoplasmosis in captive primates during the COVID-19 pandemic, identifying Toxoplasma gondii as the causative agent through a collaborative, interdisciplinary investigation.

The paper identifies two new Anelloviridae species in Callithrix penicillata using HTS and metagenomics, revealing host-virus co-diversification patterns.

This study shows that Zika virus infection during development reduces callosal area and neuron density, disrupts axon guidance, and impairs corpus callosum formation in mice.

This study shows that ZIKV infection disrupts copper homeostasis in human astrocytes, leading to oxidative stress and cell death, and that copper chelation can reduce oxidative stress and improve cell viability.

The paper reports the genomic characterization of a novel SARS-CoV-2 lineage from Rio de Janeiro, Brazil, identifying five SNVs and the E484K mutation associated with antibody escape.

This paper investigates whether saxitoxin from cyanobacteria worsens neural damage caused by Zika virus in Brazil, finding increased cell death and brain abnormalities in experimental models.

This study integrates transcriptomic, proteomic, and genomic data from CZS neonates to identify collagen gene reductions and associated polymorphisms, linking them to neuronal migration defects and CZS susceptibility.