Author

Vincent Cattoir

Centre Hospitalier Universitaire de NantesORCID 0000-0003-4026-9912Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:bacteria (other)· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Agnès Ferroni· 1 shared Anne Doléans-JordheimiD· 1 shared Aya Ghassani· 1 shared Charlotte MichauxiD· 1 shared Claire LallementiD· 1 shared Dustin R. Klein· 1 shared Émilie Cardot-MartiniD· 1 shared Eva MichaudiD· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperMicrobiology

Agnès Ferroni

Mutual co-authors in cluster

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3 papers

Recent·Most cited

MicrobiologybioRxivPreprintJul 29, 2025

Stress-Induced Iron-Sulfur Cluster Damage as a Conserved Trigger of the Stringent Response

Eva Michaud, Lorena Ricci, Claire Lallement…·Séverin Ronneau

This study identifies Fe-S cluster damage as a trigger of the stringent response in Gram-negative pathogens, linking metabolic stress to virulence and transcriptional control.

DOI 10.1101/2025.07.29.666095

experimental assaywet-lab / biochemical assay

𝕏 PostBluesky

MicrobiologyAntimicrobial Agents and ChemotherapyPeer-reviewedDec 11, 2023

Mutations in genes lpxL1, bamA and pmrB impair the susceptibility of cystic fibrosis strains of Pseudomonas aeruginosa to murepavadin

Aya Ghassani, Pauline Triponney, Maxime Bour…·Patricia Mariani‐Kurkdjian

The paper assesses murepavadin's activity against CF-related P. aeruginosa strains and identifies mutations in lpxL1, bamA, and pmrB that increase resistance.

10 citations

humanwhole-genome sequencing

𝕏 PostBluesky

MicrobiologybioRxivPreprintJan 11, 2019

Colistin heteroresistance in Enterobacter cloacae is mediated by PmrAB-independent 4-amino-4-deoxy-l-arabinose addition to lipid A

Katie N. Kang, Dustin R. Klein, Misha I. Kazi…·Joseph M. Boll

This paper identifies a PmrAB-independent mechanism of colistin heteroresistance in Enterobacter cloacae involving PhoPQEcl-mediated addition of 4-amino-4-deoxy-l-arabinose to lipid A.

DOI 10.1101/516872

bacteria (other)proteomics

𝕏 PostBluesky