Attention Deficit Hyperactivity Disorder

This study uses Swedish registry data to predict substance use disorders in ADHD youth with machine learning models, achieving moderate accuracy even without prior SUD diagnosis.

This study examines ADHD symptoms in very preterm children, finding higher rates of subthreshold ADHD and associations with cognitive and socio-demographic factors.

The paper investigates whether BOLD signal differences in ADHD during inhibitory control tasks reflect task-directed control or symptom management, finding correlations with symptoms rather than SSRT.

This study compares fixed and time-delayed feedback reference signals in biofeedback systems for children with ADD, finding variable effects based on participant characteristics.

This study identifies ADHD risk genes by linking ID-related genes to ADHD through common variants and validates their role in Drosophila behavior.

This study identifies dopaminergic signaling as a genetic link between ADHD and obesity through genome-wide analyses and brain volume associations.

This study uses ensemble learning techniques on multimodal neuroimaging data to predict adult outcomes in childhood ADHD, achieving improved classification performance for ADHD vs. controls and remitters vs. persisters.

The paper uses independent component analysis to identify reduced fronto-striatal volume in ADHD across two cohorts, showing consistent results across age and demographic differences.

The paper identifies structural brain network alterations in ADHD and their association with symptoms and cognitive function using source-based morphometry in a family cohort.

This study examines how neurofeedback affects alpha oscillations and inhibitory control in adult ADHD patients, finding that post-neurofeedback alpha normalization correlates with improved motor inhibition.

This study identifies EEG spectral power as a neuromarker for adult ADHD, while finding that theta/beta ratio is not a reliable classifier.

This study uses Mendelian randomization to investigate bidirectional causal relationships between ADHD liability and substance use, finding evidence of causal effects in both directions for smoking and cannabis use.

The paper compares brain morphology between ADHD-only and ADHD with comorbid ODD/CD, finding shared and distinct structural differences.

The paper identifies reduced structure-function coupling in ADHD, linking it to increased neural noise variability in fronto-parietal networks.

The paper investigates the functional outcomes associated with low ADHD polygenic scores, finding lower ADHD risk and better cognitive and health outcomes in individuals with the lowest scores.

This study identifies three novel genes associated with ADHD by linking genetic variants to differential DNA methylation and gene expression in brain tissues.

This study performs a transcriptome-wide association study (TWAS) of ADHD, identifying 9 significant hits and linking them to dopaminergic and norepinephrine pathways, as well as several phenotypes.

This paper identifies five irritability trajectory classes in a longitudinal cohort, linking early-onset patterns to ADHD and late-onset patterns to depression, suggesting distinct developmental origins.

The paper identifies genetic correlations between ADHD and brain volumes, finding negative associations with intracranial volume and specific brain regions.

This study investigates reinforcement learning in ADHD using fMRI, finding altered neural correlates of error processing and dopamine regulation in ADHD compared to typically developing adolescents.

The paper investigates polygenic associations between ADHD and literacy/language abilities, finding that genetic confounding with educational attainment masks ADHD-specific reading impairments.

The paper examines associations between ADHD symptoms and brain network structure in healthy adults, finding significant links between symptoms and connectivity patterns in key brain networks.

Genome-wide association meta-analysis of 20,183 ADHD cases and 35,191 controls finds 12 significant loci; signal concentrates in conserved regions, constrained genes, and CNS regulatory elements.

This study dissociates perceptual and executive deficits in ADHD using a task that varies low-level stimulus features and measures task-irrelevant motor output, finding higher perceptual variability and TIMO in ADHD participants.

This study evaluates ADHD genetic risk variants' association with general psychopathology and specific neurodevelopmental traits, finding general pleiotropy and a specific link to hyperactivity/impulsivity.

The paper identifies a dimensional biotype in medication-naïve adults with ADHD using brain-behavior patterns from functional connectivity and symptom correlations.

This study investigates genetic mechanisms underlying sex bias in ADHD prevalence using genome-wide data and finds no mean polygenic burden difference across sexes but increased risk in female siblings and comorbid conditions.