Author

A. E. Argento

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Comba· 2 shared M. G. Castro· 2 shared P. J. Dunn· 2 shared P. R. Lowenstein· 2 shared A. A. Zarouk· 1 shared A. Kahana· 1 shared B. A. Humphries· 1 shared B. M. Baker· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

A. Comba

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Fiber density and matrix stiffness modulate distinct cell migration modes in a 3D stroma mimetic composite hydrogel

H. L. Hiraki, D. L. Matera, W. Y. Wang, A. A. Zarouk, A. E. Argento, J. M. Buschhaus, B. A. Humphries, G. D. Luker, B. M. Baker

This study investigates how matrix fiber density and stiffness influence different migration modes in MCF10A cells and their apoptotic resistance.

bioRxiv · Feb 27, 2021Bioengineering

Spatiotemporal analysis of glioma heterogeneity reveals Col1A1 as an actionable 1 target to disrupt tumor mesenchymal differentiation, invasion and malignancy

A. Comba, S. M. Faisal, P. J. Dunn, A. E. Argento, T. C. Hollon, W. Al-Holou, M. L. Varela, D. B. Zamler, G. L. Quass, P. F. Apostolides, C. Abel, C. E. Brown, P. E. Kish, A. Kahana, C. G. Kleer, , P. R. Lowenstein

The paper identifies COL1A1 as a key driver of glioma mesenchymal differentiation and invasion, showing its inhibition disrupts tumor structure and improves survival.

bioRxiv · Dec 1, 2020 · PreprintCancer Biology

FYN tyrosine kinase, a downstream target of receptor tyrosine kinases, modulates anti-glioma immune responses

A. Comba, P. J. Dunn, A. E. Argento, P. Kadiyala, M. Ventosa, P. Patel, D. Zamler, F. J. Nunez, L. Zhao, M. G. Castro, P. R. Lowenstein

This study shows that FYN tyrosine kinase in glioma cells suppresses anti-glioma immune responses, and its inhibition reduces tumor growth and improves survival in immune-competent models.

bioRxiv · Dec 17, 2019Cancer Biology