Author

A. Ikeda

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Biochemistry· 1 Bioengineering· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell signaling & trafficking· 2 Research Focus:development & morphogenesis· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Organism:human· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

A. Nakaji· 1 shared C. Akazawa· 1 shared D. Hisamatsu· 1 shared E. G. Suto· 1 shared E. Kobayashi· 1 shared K. Fujimori· 1 shared K. Funato· 1 shared K. Hanaoka· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 9 mutual co-author links

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1 shared paperCell Biology

A. Nakaji

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Generation of human-pig chimeric renal organoids using iPSC technology

K. Fujimori, S. Yamanaka, K. Shimada, K. Matsui, S. Kawagoe, T. Kuroda, A. Ikeda, M. Inoue, E. Kobayashi, T. Yokoo

This study generates human-pig chimeric renal organoids using modified human-mouse chimeric methods, demonstrating successful interspecies kidney development.

bioRxiv · Mar 19, 2024Bioengineering

Membrane contact sites regulate vacuolar fission via sphingolipid metabolism

K. Hanaoka, K. Nishikawa, A. Ikeda, P. Schlarmann, S. Sasaki, S. Yamashita, A. Nakaji, S. Fujii, K. Funato

This study shows that deletion of tricalbins disrupts vacuolar morphology in yeast by altering sphingolipid metabolism through membrane contact sites.

bioRxiv · Jun 21, 2023Cell Biology

CD73-positive cell spheroid transplantation attenuates colonic atrophy

D. Hisamatsu, N. Itakura, Y. Mabuchi, R. Ozaki, E. G. Suto, Y. Naraoka, A. Ikeda, L. Ito, C. Akazawa

This study shows that transplanting CD73+ mesenchymal stem/stromal cells as spheroids reduces colonic atrophy in colitis by modulating fibroblast activity and extracellular matrix remodeling.

Pharmaceutics · Mar 4, 2023Peer-reviewedCell Biology

Lipid flippase dysfunction as a novel therapeutic target for endosomal anomalies in Alzheimer's disease

N. KANESHIRO, K. MASATO, R. IMAOKA, A. IKEDA, Y. KAMIKUBO, T. HASHIMOTO, T. IWATSUBO, T. SAKURAI, T. UEHARA, N. TAKASUGI

This study identifies lipid flippase dysfunction as a key pathogenic event in Alzheimer's disease and presents a peptide therapy to restore endosomal function.

bioRxiv · Jul 30, 2021Biochemistry