Author

A. Jaiswar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Organism:human· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Approach:genetic perturbation / CRISPR· 1 Approach:statistical model· 1

Frequent Co-Authors

D. Walerych· 2 shared J. R. Wisniewski· 2 shared M. Grochowski· 2 shared M. Kolos· 2 shared M. Lenarcik· 2 shared M. Nowak-Niezgoda· 2 shared T. Olesinski· 2 shared W. Kazmierczak· 2 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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2 shared papersCancer Biology

D. Walerych

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2 papers

Recent·Most cited

A common druggable signature of oncogenic CMYC, mutant KRAS and mutant p53 reveals functional redundancy and competition of the oncogenes in cancer

M. Grzes, A. Jaiswar, M. Grochowski, W. Wojtys, W. Kazmierczak, T. Olesinski, M. Lenarcik, M. Nowak-Niezgoda, M. Kolos, G. Canarutto, S. Piazza, J. R. Wisniewski, D. Walerych

bioRxiv · Dec 20, 2023 · PreprintCancer Biology

The response network of HSP70 defines vulnerabilities in cancer cells with the inhibited proteasome

M. Oron, M. Grochowski, A. Jaiswar, J. Legierska, K. Jastrzebski, M. Nowak-Niezgoda, M. Kolos, W. Kazmierczak, T. Olesinski, M. Lenarcik, M. Cybulska, M. Mikula, A. Zylicz, M. Miaczynska, K. Zettl, , D. Walerych

The paper identifies HSP70 as a vulnerability in cancer cells under proteasome inhibition, showing its role in suppressing drug effects and linking its expression to patient risk.

bioRxiv · Nov 16, 2021Cancer Biology