Author

A. M. Mazur

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 2 Assay:DNA methylation· 2 Organism:human· 2 Approach:genetic perturbation / CRISPR· 1 Approach:statistical model· 1

Frequent Co-Authors

A. V. Artemov· 2 shared E. B. Prokhortchouk· 2 shared S. Zhenilo· 2 shared A. Sokolov· 1 shared A. Starshin· 1 shared D. Gackowski· 1 shared D. Kaplun· 1 shared J. Szpotan· 1 shared

Activity by Year

2016-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

A. V. Artemov

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2 papers

Recent·Most cited

Cancer BiologybioRxivDec 9, 2020

An IDH-independent mechanism of DNA hypermethylation upon VHL inactivation in cancer

A. V. Artemov, S. Zhenilo, D. Kaplun…·E. B. Prokhortchouk

This study shows that VHL inactivation causes genome hypermethylation in kidney cancer cells through 2-hydroxyglutarate accumulation, independent of IDH mutations.

DOI 10.1080/15592294.2021.1971372

humanDNA methylation

𝕏 PostBluesky

Cancer BiologybioRxivDec 12, 2016

VHL inactivation without hypoxia is sufficient to achieve genome hypermethylation

A. V. Artemov, N. Zhigalova, S. Zhenilo…·E. B. Prokhortchouk

This study shows that VHL inactivation causes genome hypermethylation in normoxia, primarily at AP-1 and TRIM28 binding sites, leading to promoter hypermethylation and reduced gene expression of transcription regulators.

DOI 10.1038/s41598-018-28795-y

humanDNA methylation

𝕏 PostBluesky