Author

A. Mansell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1

Frequent Co-Authors

A. De Paoli· 1 shared B. A. Croker· 1 shared C. C. Allison· 1 shared C. Louis· 1 shared D. De Nardo· 1 shared D. Garama· 1 shared D. Gough· 1 shared D. J. Philpott· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. De Paoli

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2 papers

Recent·Most cited

STAT3 Serine phosphorylation is required for TLR4 metabolic reprogramming and IL-1β expression

J. Balic, H. Albargy, K. Luu, F. Kirby, W. Jayasekara, F. Mansell, D. Garama, N. Baschuk, D. De Nardo, C. Louis, F. Humphries, K. A. Fitzgerald, E. Latz, D. Gough, A. Mansell

This study shows that STAT3 Ser727 phosphorylation is essential for TLR4-induced metabolic reprogramming and IL-1β production in inflammation.

Nature Communications · Jul 30, 2020Peer-reviewedImmunology

NOD1 mediates non-canonical inflammasome processing of interleukin-18 in epithelial cells to Helicobacter pylori infection.

L. S. Tran, L. Ying, K. D'Costa, G. Wray-McCann, G. Kerr, L. Le, C. C. Allison, J. Ferrand, H. Chaudhry, J. Emery, A. De Paoli, S. Creed, M. Kaparakis-Liaskos, J. Como, J. Dowling, , R. L. Ferrero

This study identifies NOD1 as a key regulator of IL-18 processing in epithelial cells during Helicobacter pylori infection, independent of canonical inflammasome proteins.

bioRxiv · Mar 24, 2019 · PreprintImmunology