Author

A. Van Campenhout

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:imaging· 2 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:simulation· 1 Approach:statistical model· 1

Frequent Co-Authors

F. De Groote· 2 shared K. Desloovere· 2 shared A. Falisse· 1 shared A. Hallemans· 1 shared E. Papageorgiou· 1 shared G. Molenaers· 1 shared H. Hoang· 1 shared H. Kainz· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersPathology

F. De Groote

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2 papers

Recent·Most cited

PathologybioRxivPreprintNov 9, 2023

Identification of neural and non-neural origins of joint hyper-resistance based on a novel neuromechanical model.

J. Willaert, K. Desloovere, A. Van Campenhout…·F. De Groote

This paper develops a neuromechanical model to distinguish neural and non-neural causes of joint hyper-resistance in children with cerebral palsy using pendulum test data.

DOI 10.1101/2023.11.09.566428

humanimaging

𝕏 PostBluesky

BioengineeringbioRxivSep 14, 2019

Physics-based predictive simulations to explore the differential effects of motor control and musculoskeletal deficits on gait dysfunction in cerebral palsy: a retrospective case study

A. Falisse, L. Pitto, H. Kainz…·I. Jonkers

This study uses physics-based simulations to identify that altered muscle-tendon properties, rather than neuromuscular control or spasticity, are the main cause of crouch gait in a child with cerebral palsy.

DOI 10.1098/rsif.2019.0402

humanimaging

𝕏 PostBluesky