Author

A.-P. Morel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Assay:imaging· 1

Frequent Co-Authors

A. Puisieux· 3 shared L. Tonon· 2 shared M. Ouzounova· 2 shared A. Coutant· 1 shared A. Pinheiro· 1 shared B. Gibert· 1 shared C. Caux· 1 shared C. Degletagne· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersCancer Biology

A. Puisieux

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Spatial transcriptomics reveal pitfalls and opportunities for the detection of rare high-plasticity breast cancer subtypes

A. Coutant, V. Cockenpot, L. Muller, C. Degletagne, R. Pommier, L. Tonon, M. Ardin, M.-C. Michallet, C. Caux, M. Laurent, A.-P. Morel, P. Saintigny, A. Puisieux, M. Ouzounova, P. Martinez

The paper uses spatial transcriptomics to identify specific markers for rare high-plasticity breast cancer subtypes, showing that bulk expression signatures are limited by stromal contamination.

bioRxiv · Apr 24, 2023Cancer Biology

RAS-induced transformation of mammary epithelial cells relies on ZEB1-dependent cellular reprogramming via a paracrine process

H. De Blander, L. Tonon, F. Fauvet, R. M. Pommier, C. Lamblot, R. Benhassoun, F. Angileri, B. Gibert, M. Ouzounova, A.-P. Morel, A. Puisieux

This study shows RAS-induced transformation of mammary epithelial cells depends on ZEB1-dependent reprogramming via a paracrine process involving senescent cell-derived cytokines.

bioRxiv · Jun 23, 2022 · PreprintCancer Biology

Biophysical properties of intermediate states of EMT outperform both epithelial and mesenchymal states.

Y. Margaron, T. Nagai, L. Guyon, L. Kurzawa, A.-P. Morel, A. Pinheiro, L. Blanchoin, F. Reyal, A. Puisieux, M. Thery

The paper investigates the biophysical properties of intermediate EMT states and finds they exhibit distinct structural and mechanical features compared to epithelial and mesenchymal states, with implications for tumor-cell dissemination.

bioRxiv · Oct 8, 2019 · PreprintCell Biology