Author

B. Desvergne

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. Naldi· 1 shared A. Wilson· 1 shared B. Toffoli· 1 shared C. BLIN-WAKKACH· 1 shared C. Casals Casas· 1 shared C. Pochon· 1 shared C. Winkler· 1 shared D. MOULIN· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperPhysiology

A. Naldi

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintMar 2, 2020

Adipose-tissue derived signals control bone remodelling.

M.-B. Madel, H. Fu, D. Pierroz…·D. MOULIN

This study shows that adipose-derived signals, particularly adiponectin, regulate bone remodeling by inhibiting osteoclastogenesis and that AdipoRon can treat inflammatory bone loss.

DOI 10.1101/2020.03.02.972711

mouseexperimental assay

PhysiologybioRxivMar 25, 2019

System analysis of cross-talk between nuclear receptors reveals an opposite regulation of the cell cycle by LXR and FXR in human HepaRG liver cells

L. Wigger, C. Casals Casas, M. Baruchet…·B. Desvergne

The paper analyzes the cross-talk between nuclear receptors LXR, FXR, and PPAR in HepaRG liver cells, revealing opposite effects of FXR and LXR on cell cycle regulation and hepatocyte differentiation.

DOI 10.1371/journal.pone.0220894

humanbulk RNA-seq