Author

B. E. Talbot

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

B. J. Brown· 2 shared J. S. Schlondorff· 2 shared K. L. Boekell· 2 shared B. R. Stotter· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

4 top collaborators · 6 mutual co-author links

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2 shared papersPhysiology

B. J. Brown

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2 papers

Recent·Most cited

Gain-of-function, focal segmental glomerulosclerosis Trpc6 mutation minimally affects susceptibility to renal injury in several mouse models.

B. J. Brown, K. L. Boekell, B. R. Stotter, B. E. Talbot, J. S. Schlondorff

This study shows that a gain-of-function Trpc6 mutation in mice causes minimal susceptibility to renal injury in various models.

bioRxiv · Feb 11, 2022Physiology

Trpc6 gain-of-function disease mutation enhances phosphatidylserine exposure in murine platelets

K. L. Boekell, B. J. Brown, B. E. Talbot, J. S. Schlondorff

This study shows that a gain-of-function Trpc6 mutation enhances phosphatidylserine exposure in murine platelets, while a dominant negative mutation does not affect this process.

bioRxiv · Feb 1, 2022Cell Biology