Author

J. S. Schlondorff

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

B. E. Talbot· 2 shared B. J. Brown· 2 shared K. L. Boekell· 2 shared B. R. Stotter· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

4 top collaborators · 6 mutual co-author links

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2 shared papersPhysiology

B. E. Talbot

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2 papers

Recent·Most cited

PhysiologybioRxivFeb 11, 2022

Gain-of-function, focal segmental glomerulosclerosis Trpc6 mutation minimally affects susceptibility to renal injury in several mouse models.

B. J. Brown, K. L. Boekell, B. R. Stotter…·J. S. Schlondorff

This study shows that a gain-of-function Trpc6 mutation in mice causes minimal susceptibility to renal injury in various models.

DOI 10.1371/journal.pone.0272313

mouseexperimental assay

𝕏 PostBluesky

Cell BiologybioRxivFeb 1, 2022

Trpc6 gain-of-function disease mutation enhances phosphatidylserine exposure in murine platelets

K. L. Boekell, B. J. Brown, B. E. Talbot, J. S. Schlondorff

This study shows that a gain-of-function Trpc6 mutation enhances phosphatidylserine exposure in murine platelets, while a dominant negative mutation does not affect this process.

DOI 10.1371/journal.pone.0270431

mouseexperimental assay

𝕏 PostBluesky