Author

B. J. Pons

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:bacteria (other)· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:proteomics· 1

Frequent Co-Authors

S. van Houte· 2 shared A. Chevallereau· 1 shared A. Grasso· 1 shared A. Maestri· 1 shared A. Pettes-Duler· 1 shared C. Chong· 1 shared C. Naylies· 1 shared E. Pursey· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersMicrobiology

S. van Houte

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Author profile →

3 papers

Recent·Most cited

Bacterial defences interact synergistically by disrupting phage cooperation

A. Maestri, E. Pursey, C. Chong, B. J. Pons, S. Gandon, R. Custodio, M. Chisnall, A. Grasso, S. Paterson, K. S. Baker, S. van Houte, A. Chevallereau, E. Westra

This study identifies a novel bacterial defence system, MADS, which synergizes with CRISPR-Cas to protect against phages with anti-CRISPR genes by disrupting phage cooperation.

bioRxiv · Mar 30, 2023 · PreprintMicrobiology

Antibiotics that affect translation can antagonize phage infectivity by interfering with the deployment of counter-defences.

B. J. Pons, T. Dimitriu, E. R. Westra, S. van Houte

The paper shows that translation-inhibiting antibiotics reduce phage amplification on CRISPR-Cas bacteria by blocking phage-encoded anti-CRISPR gene expression.

bioRxiv · Sep 7, 2022 · PreprintMicrobiology

Cytolethal Distending Toxin: from mitotic DNA damage to cGAS-dependent pro-inflammatory response

B. J. Pons, A. Pettes-Duler, C. Naylies, F. Taieb, S. Hashim, S. Tadrist, Y. Lippi, G. Mirey, J. Vignard

This study identifies mitosis as a critical phase for CDT-induced DNA damage and reveals its role in triggering cGAS-dependent inflammation and cell senescence.

bioRxiv · Jun 9, 2020 · PreprintCell Biology