Author

B. MacArthur

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:flow cytometry· 1

Frequent Co-Authors

A. Petersen· 2 shared B. Preece· 2 shared H. Durden· 2 shared I. Saha· 2 shared R. Gallegos· 2 shared S. Saffarian· 2 shared W. Peppel· 2 shared E. Abu-Shah· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersBiophysics

A. Petersen

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Competitive assembly resolves the stoichiometry of essential proteins in infectious HIV-1 virions.

H. Durden, B. Preece, R. Gallegos, I. Saha, B. MacArthur, A. Petersen, W. Peppel, S. Saffarian

The paper measures the stoichiometry of Gag-Pol and gp120/gp41 proteins in infectious HIV-1 virions using a competitive assembly method, finding it similar to all released virions.

bioRxiv · Mar 10, 2024 · PreprintBiophysics

Competitive HIV budding suggests that a self-packaging gRNA:Gag-Pol complex directs HIV assembly and enforces infectivity.

H. Durden, I. Saha, B. Preece, B. MacArthur, A. Petersen, W. Peppel, R. Gallegos, S. Saffarian

The paper investigates HIV assembly by testing infectious/non-infectious genome mixtures, finding that a gRNA:Gag-Pol complex drives virion formation and infectivity.

bioRxiv · Aug 3, 2022 · PreprintMicrobiology

Fluctuations in TCR and pMHC interactions regulate T cell activation

J. R. Egan, E. Abu-Shah, O. Dushek, T. Elliott, B. MacArthur

This study introduces a stochastic model of T cell activation showing that information from TCR-pMHC interactions, not binding strength, drives immune responses and suggests optimizing stochasticity could improve therapeutics.

bioRxiv · Feb 9, 2021Immunology

Single platelet variability governs population sensitivity and initiates intrinsic heterotypic behaviours

M. S. A. Jongen, B. MacArthur, N. A. Englyst, J. West

The paper shows that single platelet variability drives population-level sensitivity and heterotypic behaviors in platelet aggregation and coagulation.

bioRxiv · Jan 22, 2020Cell Biology