Author

C. Agarinis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A. Boettcher· 1 shared A. Kostina· 1 shared B. S. Saller· 1 shared C. J. Farady· 1 shared C. J. Gross· 1 shared C. Manneville· 1 shared C. Parker· 1 shared E. Neuwirt· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

A. Boettcher

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2 papers

Recent·Most cited

Tyrosine kinase inhibitors trigger lysosomal damage-associated cell lysis to activate the NLRP3 inflammasome

E. Neuwirt, G. Magnani, T. Cikovic, A. Kostina, S. Wohrle, S. Flemming, N. J. Fischenich, B. S. Saller, O. Gorka, S. Renner, C. Agarinis, C. Parker, A. Boettcher, C. J. Farady, R. Backofen, , O. Gross

A small molecule screen identifies tyrosine kinase inhibitors that cause lysosomal damage, leading to cell lysis and NLRP3 inflammasome activation via K+ efflux.

bioRxiv · Feb 19, 2022 · PreprintImmunology

A simple and efficient CRISPR technique for protein tagging

F. Zeng, V. Beck, S. Schuierer, I. Garnier, C. Manneville, C. Agarinis, L. Morelli, L. Quinn, J. Knehr, G. Roma, F. Bassilana, M. S. Nash

The paper introduces HiTag, a CRISPR-based method for protein tagging that works in up to 66% of transfected cells with one electroporation.

bioRxiv · Apr 5, 2019Cell Biology