Author

C. Elwell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

J. Engel· 2 shared J. Sherry· 2 shared C. Herrera· 1 shared D. L. Swaney· 1 shared D. Solomon· 1 shared D. Swaney· 1 shared E. MacMahon· 1 shared E. McMahon· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersMicrobiology

J. Engel

Mutual co-authors in cluster

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2 papers

Recent·Most cited

The Chlamydia trachomatis Inc Tri1 interacts with TRAF7 to displace native TRAF7 interacting partners

C. Herrera, E. McMahon, D. L. Swaney, J. Sherry, K. Pha, K. Adams-Boone, J. Johnson, N. Krogan, M. Stevers, D. Solomon, C. Elwell, J. Engel

This paper identifies the interaction between Chlamydia trachomatis effector Tri1 and host TRAF7, showing Tri1 displaces TRAF7's native binding partners to alter its signaling during infection.

bioRxiv · Feb 26, 2024 · PreprintMicrobiology

Chlamydia trachomatis effector Dre1 interacts with dynactin to reposition host organelles during infection

J. Sherry, L. Dolat, E. MacMahon, D. Swaney, R. Bastidas, J. R. Johnson, R. Valdivia, N. J. Krogan, C. Elwell, J. Engel

This study identifies Dre1, a Chlamydia effector that interacts with dynactin to reposition host organelles during infection, enhancing bacterial fitness.

bioRxiv · Apr 15, 2022 · PreprintMicrobiology