Author

C. Kulkarni

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 2 Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Assay:metabolomics· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

E. Lavik· 2 shared J. Glaser· 2 shared L. J. Schaub· 2 shared N. Maisha· 2 shared P. S. Brookes· 2 shared V. Janeja· 2 shared A. S. Milliken· 1 shared D. Fu· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersBioengineering

E. Lavik

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Temporal Progression: A case study in Porcine Survivability through Hemostatic Nanoparticles

C. Kulkarni, N. Maisha, L. J. Schaub, J. Glaser, E. Lavik, V. Janeja

This paper analyzes the temporal progression of blood loss and cytokines in pigs treated with hemostatic nanoparticles to assess their impact on survivability.

bioRxiv · May 25, 2021 · PreprintBioengineering

Computational Design Map for Heterogeneous Experimental Studies

C. Kulkarni, N. Maisha, L. J. Schaub, J. Glaser, E. Lavik, V. Janeja

This paper develops a computational design map to identify key variables affecting hemostatic nanoparticle efficacy in trauma studies using data mining methods.

bioRxiv · May 25, 2021 · PreprintBioengineering

Complex effects of pH on ROS from mitochondrial complex II driven complex I reverse electron transport challenge its role in tissue reperfusion injury

A. S. Milliken, C. Kulkarni, P. S. Brookes

The paper investigates how pH affects ROS production via mitochondrial complex II to complex I reverse electron transport during reperfusion injury and finds that ROS generation is insensitive to pH despite changes in mitochondrial respiration.

bioRxiv · Aug 31, 2020 · PreprintBiochemistry

ALKBH7 mediates necrosis via rewiring of glyoxal metabolism

C. Kulkarni, S. M. Nadtochiy, L. Kennedy, J. Zhang, S. Chhim, H. Alwaseem, E. Murphy, D. Fu, P. S. Brookes

This study shows ALKBH7 regulates glyoxal metabolism, and its deficiency protects against necrosis and ischemia-reperfusion injury through hormetic signaling involving methylglyoxal stress.

bioRxiv · May 4, 2020Cell Biology