Author

D. Abebayehu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Bioengineering· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:host-pathogen & immune response· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

G. C. Bingham· 2 shared S. E. Ewald· 2 shared T. Barker· 2 shared A. Miller· 1 shared B. N. Pfaff· 1 shared C. M. Saunders· 1 shared C.-R. Yeh· 1 shared D. R. Griffin· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBioengineering

G. C. Bingham

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A unique Thy-1-negative immuno-fibroblast population emerges as a key determinant of fibrotic outcomes to biomaterials

D. Abebayehu, B. N. Pfaff, G. C. Bingham, S. Ghatti, A. Miller, D. R. Griffin, T. Barker

The paper identifies a Thy-1-negative fibroblast subpopulation that drives fibrosis in response to biomaterials using single-cell RNA sequencing.

bioRxiv · Jan 9, 2022Bioengineering

Myeloid-mediated IL-1R signaling in immuno-responsive Thy-1 negative fibroblasts is critical for pulmonary fibrosis

D. Abebayehu, C.-R. Yeh, G. C. Bingham, S. E. Ewald, T. H. Barker

This study identifies Thy-1 negative fibroblasts as immuno-responsive cells that link inflammation and mechanotransduction in pulmonary fibrosis through IL-1R signaling.

bioRxiv · May 11, 2021 · PreprintCell Biology

Cachexia and fibrosis are costs of chronic IL-1R-mediated disease tolerance in T. gondii infection

S. E. Ewald, S. J. Melchor, J. A. Hatter, E. A. LaTorre Castillo, C. M. Saunders, K. A. Byrnes, I. Sanders, D. Abebayehu, T. Barker, S. Countermarsh-Ott

This study shows that IL-1R signaling protects mice from acute T. gondii infection but leads to chronic cachexia and fibrosis when sustained.

bioRxiv · Sep 26, 2019 · PreprintImmunology