Author

T. Barker

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Research Focus:differential expression· 3 Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Approach:statistical model· 2

Frequent Co-Authors

A. Miller· 2 shared D. Abebayehu· 2 shared A. E. Miller· 1 shared A. Henderson· 1 shared B. N. Pfaff· 1 shared C. J. Cook· 1 shared C. M. Saunders· 1 shared C. Sano· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 10 mutual co-author links

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2 shared papersBioengineering

A. Miller

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Phylodynamics Uncovers the Transmission of Antibiotic-Resistant Escherichia coli between Canines and Humans in an Urban Environment

N. Walas, N. F. Müller, E. Parker, A. Henderson, D. Capone, J. Brown, T. Barker, J. P. Graham

This study uses genomic sequencing and phylogenetics to show antibiotic-resistant E. coli is transmitted between canines and humans in urban environments.

bioRxiv · Jun 1, 2023 · PreprintMicrobiology

Characterizing the extracellular matrix transcriptome of cervical, endometrial, anduterine cancers

C. J. Cook, A. E. Miller, T. Barker, Y. Di, K. Fogg

This study characterizes the matrisome gene expression in cervical, endometrial, and uterine cancers using TCGA and GTEx data, revealing distinct patterns linked to tumor progression and patient survival.

bioRxiv · Apr 4, 2022Cancer Biology

A unique Thy-1-negative immuno-fibroblast population emerges as a key determinant of fibrotic outcomes to biomaterials

D. Abebayehu, B. N. Pfaff, G. C. Bingham, S. Ghatti, A. Miller, D. R. Griffin, T. Barker

The paper identifies a Thy-1-negative fibroblast subpopulation that drives fibrosis in response to biomaterials using single-cell RNA sequencing.

bioRxiv · Jan 9, 2022Bioengineering

Extracellular Matrix Remodeling Associated With Bleomycin-Induced Lung Injury Supports Pericyte-To-Myofibroblast Transition

R. HANNAN, A. Miller, R.-C. Hung, C. Sano, S. M. Peirce, T. Barker

This paper investigates how pericytes transition into myofibroblasts during lung injury, showing that fibronectin and vβ3 integrin engagement drive this process and may be targeted to reduce fibrosis.

bioRxiv · Nov 16, 2020Cell Biology

Cachexia and fibrosis are costs of chronic IL-1R-mediated disease tolerance in T. gondii infection

S. E. Ewald, S. J. Melchor, J. A. Hatter, E. A. LaTorre Castillo, C. M. Saunders, K. A. Byrnes, I. Sanders, D. Abebayehu, T. Barker, S. Countermarsh-Ott

This study shows that IL-1R signaling protects mice from acute T. gondii infection but leads to chronic cachexia and fibrosis when sustained.

bioRxiv · Sep 26, 2019 · PreprintImmunology