Author

D. Agalliu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Arac· 1 shared A. Cottarelli· 1 shared A. Edinger· 1 shared A. Eichmann· 1 shared A. Snead· 1 shared A. Sproul· 1 shared B. Nelson· 1 shared B. Tavitian· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCell Biology

A. Arac

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Rab7a is required to degrade select blood-brain barrier junctional proteins after ischemic stroke

A. Cottarelli, D. Jamoul, M. C. Tuohy, S. Shahriar, M. Glendinning, G. Prochilo, A. Edinger, A. Arac, D. Agalliu

This study shows Rab7a deletion reduces BBB dysfunction after ischemic stroke by preventing degradation of junctional proteins.

bioRxiv · Aug 29, 2023 · PreprintCell Biology

Endothelial Unc5B controls blood-brain barrier integrity

K. Boye, L. H. M. Geraldo, J. furtado, L. Pibouin-fragner, M. Poulet, D. Kim, B. Nelson, Y. Xu, L. Jacob, N. Maissa, B. Tavitian, D. Agalliu, L. Claesson-Welsh, S. Ackerman, A. Eichmann

This study shows that endothelial Unc5B maintains blood-brain barrier integrity via Wnt/{beta}-catenin signaling, with potential therapeutic applications for CNS diseases.

bioRxiv · May 6, 2021Cell Biology

Human induced pluripotent stem cell-derived neuroectodermal epithelial cells mistaken for blood-brain barrier-forming endothelial cells

T. M. Lu, D. Redmond, T. Magdeldin, D. T. Nguyen, A. Snead, A. Sproul, J. Xiang, H. A. Fine, K. Shido, Z. Rosenwaks, S. Rafii, D. Agalliu, R. Lis

The paper identifies human iBMECs as EpCAM+ neuroectodermal epithelial cells rather than BBB-forming endothelial cells, showing that ETS TFs can reprogram them toward a vascular BBB in vitro.

bioRxiv · Jul 11, 2019Cell Biology