Author

D. Beeson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:protein folding & dynamics· 1 Assay:proteomics· 1 Assay:structural biology· 1

Frequent Co-Authors

A. C. Pike· 1 shared A. Chu· 1 shared A. Sajid· 1 shared A. Vincent· 1 shared B. G. Davis· 1 shared C. E. Barry· 1 shared C. V. Robinson· 1 shared D. A. Widdick· 1 shared

Activity by Year

2018-2018 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperBiochemistry

A. C. Pike

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Isolation and characterization of rare circulating autoantibody-producing cells from patients with muscle-specific kinase myasthenia gravis

K. Takata, P. Stathopoulos, M. Cao, M. Mane-Damas, M. Fichtner, E. S. Benotti, L. Jacobson, P. Waters, S. R. Irani, P. Martinez-Martinez, D. Beeson, M. Losen, A. Vincent, R. J. Nowak, K. C. O'Connor

The paper isolates and characterizes MuSK-specific autoantibody-producing B cells from MuSK myasthenia gravis patients, generating monoclonal antibodies that reveal distinct binding specificities and functional effects on AChR clustering.

bioRxiv · Dec 13, 2018Immunology

Structures of DPAGT1 explain glycosylation disease mechanisms and advance TB antibiotic design

Y. Y. Dong, H. Wang, A. C. Pike, S. J. Cochrane, S. Hamedzade, F. J. Wyszynsk, S. R. Bushell, S. F. Royer, D. A. Widdick, A. Sajid, H. I. Boshoff, R. Lucas, W.-M. Liu, S. S. Lee, T. Machida, , E. P. Carpenter

This paper determines the structures of DPAGT1 with substrates and inhibitors, revealing catalytic mechanisms and disease-causing mutations, and designs non-toxic antibiotics for tuberculosis.

bioRxiv · Apr 2, 2018Biochemistry