Author

D. Diez

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Assay:scRNA-seq· 2 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

D. M. Standley· 2 shared A. Tanaka· 1 shared E. L. Lim· 1 shared G. L. Abe· 1 shared J.-I. Sasaki· 1 shared M. A. Llamas-Covarrubias· 1 shared M. d. J. Loza Lopez· 1 shared M. Loza· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

D. M. Standley

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2 papers

Recent·Most cited

The TCRs of pathogenic Th17 cells in arthritogenic mice are shifted toward a Treg-like repertoire

M. A. Llamas-Covarrubias, A. Tanaka, M. d. J. Loza Lopez, D. Diez, Z. Xu, E. L. Lim, S. Teraguchi, S. Sakaguchi, D. M. Standley

The paper shows that ZAP-70 mutations in mice lead to a shift in T cell repertoires, with pathogenic Th17 cells resembling Tregs and contributing to autoimmune arthritis.

bioRxiv · Mar 1, 2024 · PreprintImmunology

Intercellular crosstalk in adult dental pulp is mediated by heparin-binding growth factors Pleiotrophin and Midkine

N. Jiravejchakul, G. L. Abe, M. Loza, S. Park, P. Matangkasombut, J.-I. Sasaki, S. Imazato, D. Diez, D. M. Standley

The paper analyzes single-cell transcriptomes of dental pulp to identify unique gene expression profiles and growth factor-mediated crosstalk between cell types.

bioRxiv · Oct 28, 2022Cell Biology