Author

D. E. Kahn

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. D'Alessandro· 1 shared A. L. Rachubinski· 1 shared H. C. Lewis· 1 shared J. C. Costello· 1 shared J. M. Espinosa· 1 shared K. A. Waugh· 1 shared K. D. Sullivan· 1 shared K. D. Tuttle· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

A. D'Alessandro

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1 paper

Recent·Most cited

Trisomy 21 activates the kynurenine pathway via increased dosage of interferon receptors

R. K. Powers, K. D. Sullivan, R. Culp-Hill, M. P. Ludwig, K. P. Smith, K. A. Waugh, R. Minter, K. D. Tuttle, H. C. Lewis, A. L. Rachubinski, R. E. Granrath, R. B. Wilkerson, D. E. Kahn, M. Joshi, A. D'Alessandro, , J. M. Espinosa

This study identifies a mechanism by which Trisomy 21 increases kynurenine pathway activity through overexpression of interferon receptors, leading to immunosuppression and neurotoxicity in Down syndrome.

bioRxiv · Jan 11, 2019Physiology