Author

D. Kadekar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

J. Rizk· 2 shared M. T. Vinals· 2 shared R. Agerholm· 2 shared V. Bekiaris· 2 shared A. Taibi· 1 shared B. Maurer· 1 shared E. C. Tardos· 1 shared E. Comelli· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersImmunology

J. Rizk

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Author profile →

2 papers

Recent·Most cited

The cIAP ubiquitin ligases sustain type 3 γδ T and innate lymphoid cells during aging to allow normal cutaneous and mucosal responses

J. Rizk, U. M. Morbe, R. Agerholm, I. Ulmert, E. C. Tardos, D. Kadekar, M. V. Baglioni, M. T. Vinals, V. Bekiaris

This study shows that cIAP1/2 ubiquitin ligases are essential in early life for maintaining IL-17-producing γδT17 and ILC3 cells, which are critical for immune responses during aging.

bioRxiv · May 14, 2020Immunology

The neonatal microenvironment programs conventional and intestinal Tbet+ γδT17 cells through the transcription factor STAT5

D. Kadekar, R. Agerholm, J. Rizk, H. Neubauer, T. Suske, B. Maurer, M. T. Vinals, E. Comelli, A. Taibi, R. Moriggl, V. Bekiaris

This study shows STAT5 is critical for neonatal expansion of γδT17 cells, which acquire tissue-specific traits necessary for immunity and autoimmunity.

bioRxiv · Jun 3, 2019Immunology