Author

D. Pally

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 4

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 4 Approach:experimental assay· 3 Assay:proteomics· 3 Research Focus:cell signaling & trafficking· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

R. Bhat· 4 shared D. Pramanik· 2 shared R. V. Kumar· 2 shared S. Hussain· 2 shared A. Balakrishnan· 1 shared A. Chatterjee· 1 shared A. Petersson· 1 shared A. Srinivas· 1 shared

Activity by Year

2018-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

4 shared papersCancer Biology

R. Bhat

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Galectin-9 signaling drives breast cancer invasion through matrix

D. Pally, M. Banerjee, S. Hussain, R. V. Kumar, A. Petersson, E. Rosendal, L. Gunnarsson, K. Peterson, H. Leffler, U. J. Nilsson, R. Bhat

This study shows Galectin-9 promotes breast cancer invasion through ECM by enhancing S100A4 expression and FAK signaling.

bioRxiv · Nov 6, 2021 · PreprintCancer Biology

ST6GAL1-mediated heterogeneity in sialic acid levels potentiates invasion of breast cancer epithelia

D. Pally, D. Pramanik, S. Hussain, S. Verma, A. Srinivas, R. V. Kumar, R. Bhat

This study identifies 2,6-linked sialic acid heterogeneity in breast cancer cells and shows that medium-expressing cells exhibit greater ECM adhesion and invasion compared to high-expressing cells, with ST6GAL1 playing a key role.

ACS Central Science · Jan 4, 2021Peer-reviewedCancer Biology

An interplay between reaction-diffusion and cell-matrix adhesion regulates multiscale invasion in early breast carcinomatosis

D. Pally, D. Pramanik, R. Bhat

This paper introduces a multiscale computational model to study how reaction-diffusion and cell-matrix adhesion regulate breast cancer invasion.

bioRxiv · Mar 4, 2019Cancer Biology

Galectin-1 promotes the invasion of bladder cancer urothelia through their matrix milieu

A. Balakrishnan, D. Pally, K. Gondkar, S. Naito, D. Sidransky, A. Chatterjee, P. Kumar, R. Bhat

This study identifies Galectin-1 as a driver of bladder cancer invasion through matrix interactions, showing its role in promoting adhesion, proliferation, and mesenchymal-like phenotypes.

bioRxiv · Oct 12, 2018 · PreprintCancer Biology